Precision of computed spectrophotometric scans for heme pigments in subarachnoid hemorrhage makes interpretation reliable.

نویسندگان

  • Daniel M O'Connell
  • Ian D Watson
چکیده

References 1. Bell GI, Polonsky KS. Diabetes mellitus and genetically programmed defects in -cell function. Nature 2001;414:788–91. 2. Gabir MM, Hanson RL, Dabelea D, Imperatore G, Roumain J, Bennett PH, et al. Plasma glucose and prediction of microvascular disease and mortality: evaluation of 1997 American Diabetes Association and 1999 World Health Organization criteria for diagnosis of diabetes. Diabetes Care 2000;23:1113–8. 3. Stride A, Hattersley AT. Different genes, different diabetes: lessons from maturity-onset diabetes of the young. Ann Med 2002;34:207– 16. 4. Davis EA, Cuesta-Munoz A, Raoul M, Buettger C, Sweet I, Moates M, et al. Mutants of glucokinase cause hypoglycaemiaand hyperglycaemia syndromes and their analysis illuminates fundamental quantitative concepts of glucose homeostasis. Diabetologia 1999;42:1175–86. 5. Bonora E, Targher G, Alberiche M, Formentini G, Calcaterra F, Lombardi S, et al. Predictors of insulin sensitivity in type 2 diabetes mellitus. Diabet Med 2002;19:535–42. 6. Lindner TH, Cockburn BN, Bell GI. Molecular genetics of MODY in Germany. Diabetologia 1999;42:121–3. 7. Ferre T, Riu E, Franckhauser S, Agudo J, Bosch F. Long-term overexpression of glucokinase in the liver of transgenic mice leads to insulin resistance. Diabetologia 2003;46:1662–8. 8. Monti LD, Landoni C, Setola E, Galluccio E, Lucotti P, Sandoli EP, et al. Myocardial insulin resistance associated with chronic hypertriglyceridemia and increased FFA levels in type 2 diabetic patients. Am J Physiol Heart Circ Physiol 2004;287:H1225–31. 9. Temelkova-Kurktschiev T, Hanefeld M. The lipid triad in type 2 diabetes—prevalence and relevance of hypertriglyceridaemia/low high-density lipoprotein syndrome in type 2 diabetes. Exp Clin Endocrinol Diabetes 2004;112:75–9. 10. Palmer AM, Murphy N, Graham A. Triglyceriderich lipoproteins inhibit cholesterol efflux to apolipoprotein (apo) A1 from human macrophage foam cells. Atherosclerosis 2004;173: 27–38.

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عنوان ژورنال:
  • Clinical chemistry

دوره 51 4  شماره 

صفحات  -

تاریخ انتشار 2005